By Alan G. Rose
This atlas offers a finished representation and outline of a variety and variety of pathologic tactics and ailments affecting all of the significant organs of the physique. Emphasis is put on how the anatomic constitution of other organs may well ascertain the development of involvement by way of sickness approaches and the way such styles might relief within the right prognosis of the gross pathology. from time to time, a number of illustrations of disorder strategies are given to teach evolution of the sickness. Histologic illustrations of chosen gross lesions also are integrated the place correct. The atlas is illustrated with greater than 1,200 colour photomicrographs. Atlas of Gross Pathology is vital for working towards pathologists, yet may also supply a great origin for surgeons, internists, and obstetricians/gynecologists.
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Additional info for Atlas of Gross Pathology: With Histologic Correlation
Histology of acute myocardial infarction. A. Edge of an infarct of about 6 hours duration showing coagulative necrosis (left) and damaged, viable myocytes (right). B. Edge of expanding infarct of about 12 hours duration showing contraction band necrosis of myocytes, interstitial edema, plus a prominent polymorphonuclear leukocytic infiltration. C. Central portion of a myocardial infarct of about 4 days duration showing neutrophils infiltrating between myocytes showing advanced coagulative necrosis.
A. Cardiac aneurysm of left ventricular apex complicating myocardial infarction. B. Remnants of the original myocardial wall in the wall of the aneurysm signify the presence of a true cardiac aneurysm. qxd 5/12/08 12:22 PM Page 45 A 1-72. A. Transverse section of ventricles showing focal hemorrhage within the left ventricle due to a reperfused myocardial infarct. A large intraepicardial hematoma is present over the right ventricle, and death occurred due to massive hemopericardium (cardiac tamponade) that clinically mimicked cardiac rupture.
B. Opened mitral valve showing multiple redundant folds (hoods) on posterior leaflet prolapsing back into the left atrium. Intervening portions of the cusp appear normal. C. Hoods develop at sites of absent fibrosal collagen and expanded spongiosa (loose connective tissue rich in acid mucopolysaccharide). qxd 5/12/08 D 12:21 PM Page 29 1-43. (Continued) D. Endocardial friction lesion of endocardium behind chordae. E. Histologic appearance of the friction lesion showing red staining collagen layer expanding the endocardium.
Atlas of Gross Pathology: With Histologic Correlation by Alan G. Rose